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Industrial toxic peripheral neuropathy

Toxic peripheral neuropathy caused by industrial toxicants, which takes the peripheral nerve as the main target organ, includes acrylamide, n-hexane, carbon disulfide, trichloroethylene, chloropropene, methyl-n-butyl ketone, ethylene oxide, lead, arsenic and thallium, as well as some organic phosphate compounds, such as trichlorfon, dichlorvos, methamidophos, parathion, dimethoate, Omethoate, malathion, profluorophos and TOCP; Peripheral neuropathy with single nerve damage can occur in acute carbon monoxide poisoning. Peripheral neuropathy caused by industrial poisons is mostly manifested as polyneuropathy in clinic. Generally, the disease starts covertly and develops gradually, which is characterized by the decline or disappearance of symmetrical sensory, motor and tendon reflex at the distal end of the limb, or accompanied by autonomic nerve dysfunction. The basic histopathological changes of peripheral neuropathy mainly include Wallerian degeneration, axonal degeneration, segmental demyelination and axonal atrophy.

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